Turan, BetulGoktas, EmineZamani, Ayse GulTokgoz, HuseyinYildirim, Mahmut Selman2024-02-232024-02-2320222147-2092https://doi.org/10.12996/gmj.2022.68https://hdl.handle.net/20.500.12452/15186Chromosome 1q gain that confers clonal expansion advantage to tumor cells has been reported in many solid tissue and hematological cancers, in many different forms; sometimes as a derivative chromosome, as isochromosome, or less frequently, due to an imbalance created by a jumping translocation. Although it is known that chromosome 1q gain provide the advantage of clonal expansion to the tumor cells and is relatively common in Burkitt lymphoma/leukemia, its detection in the form of jumping translocation is extraordinarily rare and results of JT containing 1q are controversial. Bone marrow cytogenetic examination performed on a case diagnosed with stage 4 Burkitt lymphoma/leukemia resulted in 46,XY,dup(1)(q21q42),t(8;14)(q24;q32)[5]/46,XY,der(6)t(1;6)(q21;q27),t(8;14)(q 24;q32)[4]/46,XY,t(8;14)(q24;q32), der(11)t(1;11)(q21;q23 )[2]/46,XY[3]. We present the clinical features of the case that was found to have 1q gain in the jumping translocation form to contribute to the literature.eninfo:eu-repo/semantics/openAccessBurkitt's LeukemiaBurkitt's LymphomaBone MarrowCytogenetic AbnormalitiesTranslocationChromosomalArticle3332912932-s2.0-85133690631Q4WOS:00082315140002010.12996/gmj.2022.68