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    Pseudoaneurysm originating from left ventricle aneurysm: An autopsy case and review of literature
    (Elsevier Sci Ltd, 2013) Dogan, Kamil Hakan; Demirci, Serafettin; Tavli, Lema; Buken, Bora
    Rupture of the free wall of the left ventricle is a catastrophic complication of acute myocardial infarction. Rarely, free wall rupture is contained by overlying adherent pericardium, producing a pseudoaneurysm of the left ventricle. In this report, a case of a left ventricular pseudoaneurysm due to a previous myocardial infarction is described. A 55-year-old woman had a severe chest pain 11 months prior to death. No cardiac investigation was performed. Three days prior to death, she suffered from fatigue and weakness, and had a witnessed sudden cardiac death. At autopsy, a 8.5 x 10 x 8 cm pseudoaneurysm of the left ventricle was found. There was severe coronary artery atherosclerosis. There were extensive adhesions between pericardium and pseudoaneurysm wall. The cause of death was attributed to heart failure and resulting arrhythmia. The case illustrates the rare event of left ventricular pseudoaneurysm first diagnosed at forensic autopsy. (C) 2013 Elsevier Ltd and Faculty of Forensic and Legal Medicine. All rights reserved.
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    Suicide in two patients with epilepsy and cystic degeneration areas of their brains
    (Romanian Legal Med Soc, 2013) Dogan, Kamil Hakan; Demirci, Serafettin; Baba, Z. Fusun; Buken, Bora
    Suicide is an important cause of death among patients with epilepsy. In this report, we describe two patients with epilepsy whose brain examinations revealed cystic degeneration areas. The first case was a 42-year-old man who had been treated for epilepsy for five years. It was reported that his epilepsy attacks had increased over the six months prior to his suicide, and that he had severe conflicts with his wife during the last two months. He committed suicide by hanging. An autopsy revealed two cystic degeneration areas on the left temporal lobe, and one cystic degeneration area on the right parietal lobe. The second case was a 30-year-old man who had been receiving epilepsy treatment for 10 years. It was reported that his epilepsy attacks had increased in the four months prior to his suicide, and his wife had left home because of continuous arguments. He committed suicide by drinking rodenticide (aluminum phosphide). An autopsy revealed a cystic degeneration area on the right temporal lobe. Neither of the two cases had a history of head injury, and the cystic areas are evaluated as border zone infarcts due to prolonged hypoxemia caused by recurrent status epilepticus attacks. In conclusion, in case of the determination of the cystic degeneration areas in patients with epilepsy in computed tomography or magnetic resonance imaging examinations, it will be beneficial to follow these patients in terms of epileptic attacks and psychiatric defects.