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    Preventive Effects of Intraperitoneal Selenium on Cerebral Vasospasm In Experimental Subarachnoid Hemorrhage
    (Lippincott Williams & Wilkins, 2010) Kocaogullar, Yalcin; Ilik, Kemal; Esen, Hasan; Koc, Osman; Guney, Onder
    Vasospasm is an important cause of morbidity and mortality with subarachnoid hemorrhage (SAH). The effect of intraperitoneal administration of selenium, which is an antioxidant on cerebral vasospasm was investigated in an experimental model. By means of intracisternal blood injection model, SAH was induced in 24 rabbits, which were randomly divided into 3 groups (group I = control group, group 2 = SAH alone group, and group 3 = SAH plus selenium group). Basilar artery angiography was performed on day 0 and day 3 as described. Intraperitoneal selenium (0.05 mg/kg) treatment was started after the induction of SAH and administered once a day. Three days later, the animals were killed and the basilar artery was examined histologically for the luminal diameter and thickness of the arterial muscular wall. The mean values for the measurements of angiographic luminal diameter, pathologic luminal area.. Muscular wall thickness derived from the blind observer were analyzed statistically. There was no statistically significant difference in basal angiographic luminal diameter evaluation between groups 1-2-3 (P > 0.005). But in third day angiography; comparison of Group 2 and group 1-3 showed statistically significant differences (P < 0.001). In pathologic investigation; there was statistically significant difference in luminal area and muscular wall thickness of the basilar artery between groups 1, 2, and 3 (P < 0.005). Intraperitoneal selenium treatment was found effective by increasing the angiographic diameter, pathologic luminal area and reducine muscular wall thickness measurements. This is the first study to show that intraperitoneal administration of selenium is effective in preventing vasospasm after SAH in rabbits.
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    Varicella Zoster of the Trigeminal Nerve After Radiofrequency Thermocoagulation of the Gasserian Ganglion: A Case Report
    (Lippincott Williams & Wilkins, 2013) Ilik, Kemal; Erdi, Fatih; Ilik, Faik; Keskin, Fatih
    In this report we present a case of varicella zoster infection at the third division of trigeminal nerve, which was developed after radiofrequency thermocoagulation (RFT) of the Gasserian ganglion for the treatment of trigeminal neuralgia. A-65-year-old female patient who was suffering from left-sided facial pain, a vesicular erythematous rash in the mandibular division of the trigeminal nerve, and left eye pain, was admitted to our department. The patient had suffered from trigeminal neuralgia for 5 years. Her complaints increased during the past 6 months and became refractory to the medical treatment. Finally, the patient underwent RFT of the Gasserian ganglion 1 month ago. During the past 10 days, vesicular erythematous rash, and left eye pain and tearing occurred with an exacerbation of left-sided facial pain. The painful rash consisted of varicella zoster infection. This is the first report about reactivation of varicella zoster infection after RFT in the literature. RFT of Gasserian ganglion might act as a precipitator in the reactivation of latent varicella zoster infection.